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Cerebral Cortex

Neurodegenerative and Age-Related Changes in Structure and Function of Cerebral Cortex

Inbunden, Engelska, 1999

AvAlan Peters,John H. Morrison,John H Morrison

4 199 kr

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This volume is concerned with neurodegenerative disorders and age-related changes in the structure and function of the cerebral cortex, a topic that has attracted increasing interest as longevity and the number of aged individuals in the population increase.

Produktinformation

Utgivningsdatum1999-09-30
Mått178 x 254 x 43 mm
Vikt1 619 g
FormatInbunden
SpråkEngelska
SerieCerebral Cortex
Antal sidor783
Upplaga1999
FörlagSpringer Science+Business Media
ISBN9780306459665

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1 Cognitive Profiles of Normal Human Aging.- 2 Age-Related Cognitive Decline in the Rhesus Monkey.- 3 Normal Aging in the Cerebral Cortex of Primates.- 4 Age-Related Neuronal Loss in the Cerebral Cortex.- 5 Age-Related Cognitive Deficits and Neurotransmitters: The Role of Catecholamine Mechanisms in Prefrontal Cortical Cognitive Decline.- 6 Neurotransmitter Receptor Changes in the Hippocampus and Cerebral Cortex in Normal Aging.- 7 Trophic Factors in Experimental Models of Adult Central Nervous System Injury.- 8 Cortical Neuropathology in Aging and Dementing Disorders: Neuronal Typology, Connectivity, and Selective Vulnerability.- 9 The Anatomy of Dementias.- 10 Age-Related Changes in Subcortical Nuclei that Project to the Cerebral Cortex.- 11 Ultrastructural Changes in Dementing Illnesses.- 12 Locating Genetic Modifiers for Inherited Neurodegenerative Diseases.- 13 Early Features of Alzheimer’s Disease.- 14 Temporal Sequence of Alzheimer’s Disease-Related Pathology.- 15 Neuropathological Correlates of Dementia in Alzheimer’s Disease.- 16 Multifocal Cortical Neurodegeneration in Alzheimer’s Disease.- 17 Molecular Pathology of Alzheimer’s Disease and Related Disorders.- 18 Glutamate Receptors and Excitotoxic Mechanisms in Alzheimer’s Disease.- 19 Nerve Growth Factor Systems in Alzheimer’s Disease.- 20 Vascular-Related and Mediated Alterations in Alzheimer’s Disease.